紫外线（UV）处理已被证明会产生几乎无法检测到的辐射水平 常用剂量的消毒副产物（DBPs）。大多数紫外线处理 方案包括氯化或氯胺化，以维持 分发过程中残留的消毒剂。出于这个原因，人们也对 紫外线处理可能对天然有机物（NOM）或DBP前体产生影响。 几个研究小组已经研究了这个问题，但迄今为止的研究结果未能证明这一点 在典型的紫外线剂量下表现出显著的效果。 这项研究的目的是扩展之前的工作，将研究包括在内 总有机卤化物（Tox）及短时THM和HAa地层的研究 潜伏期。来自不同化学和地理位置的原水 收集光源，并在实验室中用紫外线（低压）和氯进行处理 麻省大学实验室。注意使用实际的氯剂量（2-6 mg/L）和 控制pH值，以及中高紫外线剂量（400-1500 mJ/cm²）。 结果表明，THMs和HAAs的形成率显著增加 当水被注入典型水平的紫外线辐射时。这表现为8- 两个受调节的DBP组在短氯接触时间内增加23%。先前的 研究只考虑了较长的反应时间，这可能解释了为什么这些影响 之前没有报道过。此外，TOX的数量也大幅增加 由于紫外线预处理。大约30分钟后，这些增加的幅度在85-270%之间 几分钟的联系时间。从这些数据中可以明显看出，紫外线处理具有 对DPB前体的影响比之前认识到的要大得多。 它似乎也 产生不同分布的DBP，有利于非THM、非HAA副产物。 包括17个参考文献、表格、图表。

Ultraviolet (UV) treatment has been shown to produce a nearly undetectable level of disinfection byproducts (DBPs) at the doses commonly used. Most UV treatment scenarios include either chlorination or chloramination for the purpose of maintaining a disinfectant residual during distribution. For this reason, there has also been an interest in impacts UV treatment might have on natural organic matter (NOM) or DBP precursors. Several research groups have examined this question, and results to date have failed to show a substantial effect at typical UV doses. The purpose of this research was to expand upon the prior work to include studies of total organic halide (TOX) and to consider THM and HAA formation under short incubation times. Raw waters from several chemically and geographically distinct sources were collected and treated with UV light (low pressure) and chlorine in the UMass laboratory. Care was taken to use realistic chlorine doses (2-6 mg/L) and controlled pH, along with medium to high UV doses (400-1500 mJ/cm²). Results showed substantial increases in rate of formation of THMs and HAAs when waters were dosed with typical levels of UV radiation. This manifested itself as 8- 23% increases in the two regulated DBP groups at short chlorine contact times. Prior studies have only considered long reaction times, which might explain why these effects have not previously been reported. In addition, there were substantial increases in TOX as a result of UV pretreatment. These increases ranged from 85-270% after about 30 minutes of contact time. What is apparent from this data is that UV treatment has a much greater influence on DPB precursors than previously recognized. It also appears to produce a different distribution of DBPs, favoring the non-THM, non-HAA byproducts. Includes 17 references, tables, figures.