研究发现，患者走廊中的气流是几例空气传播疾病医院内传播的促因。结果，一家实际医院被用来观察在中性（非定向）和负（定向）气流下普通病房两条走廊中合成呼吸气溶胶的空气动力学行为。在负模式下，与中性模式（13.2米，43.3英尺）相比，1.0-3.0微米的气溶胶保持在背景浓度以上，距离注入点的距离（28.4米，93.2英尺）是后者的两倍多，这表明扩散可能是1.5微米的重要传输机制。 0微米和3.0微米颗粒。相比之下，0.5μm气溶胶在超过31.5m（103.3ft）的距离内仍高于背景浓度，而气流模式相对较少，这表明扩散可能是0.5μm颗粒的重要传输机制。因此，数据表明，与0.5μm颗粒（例如病毒滴核）相比，患者走廊内的定向气流可能对1.0μm和3.0μm呼吸颗粒（例如细菌和真菌）的运动产生更大的影响。引文:ASHRAE论文CD:2014 ASHRAE年会，华盛顿州西雅图

Airflow in patient corridors was found to be a contributing factor in several cases of nosocomial transmission of airborne disease. As a result, an actual hospital was used to observe the aerodynamic behavior of synthetic respiratory aerosols in two corridors of a general patient ward placed under neutral (non-directional) and negative (directional) airflow. Under the negative mode, aerosols 1.0-3.0 microm remained above background concentrations more than twice the distance (28.4m, 93.2ft) from the injection point when compared to the neutral mode (13.2m, 43.3ft), suggesting that dispersion may be a significant transport mechanism for 1.0 microm and 3.0 microm particles. In contrast, 0.5μm aerosols remained above background concentrations to distances exceeding 31.5m (103.3ft) with comparatively less regard to airflow mode, suggesting that diffusion may be a significant transport mechanism for 0.5μm particles. As a result, data indicate that directional airflow within a patient corridor may have a greater effect on the movement of 1.0μm and 3.0μm respiratory particles (e.g. bacteria and fungi) compared to 0.5μm particles (e.g. viral droplet nuclei).